Why does venous block cause infarction?

In summary, the conversation discusses the difference between blocked arteries and blocked veins and how it leads to necrosis of tissue. It is explained that when a vein is blocked, it causes congestion and increased back pressure, making it difficult for arterial blood to come and perfuse tissue. The conversation also asks for further clarification on the pathogenesis of cerebral venous infarction and the occurrence of haemorrhagic infarct. It is suggested that this can be understood as a balance between blood flow in and out, as prolonged blood staying in one area can decrease oxygen transfer to the tissue.
  • #1
sameeralord
662
3
Hi guys,

I know this is a simple question. But when an artery is blocked it is easier to understand because, less oxygen goes to tissue so, necrosis of tissue. But when a vein is blocked, only the draining is affected, the arteries are still able to supply oxygen, but why does necrosis still occur. I'm assuming it is because when a vein is blocked, it leads to congestion, increased back pressure, which makes it difficult for arterial blood to come and perfuse tissue. Just asked to clarify (I know this is probably a pretty obvious stupid question, but I like to get it clarified). Thanks :smile:

While we are at it, if you know something about this, please tell me

Does the pathogenesis of cerebral venous infarction differ considerably from the pathogenesis of cerebral arterial ischemic infarction. Why does a haemorrhagic infarct occur in cerbral venous infarction.
 
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  • #2
You can simply see this as "what flows in must flow out", because if that is not the case some volume of blood will stay in the same area for a prolonged time and the transfer of oxygen to the tissue from this volume of blood will go down as their oxygen levels get closer together.
 

1. Why does venous block cause infarction?

Venous block causes infarction because it restricts the flow of blood and oxygen to a particular area of tissue. This lack of oxygen can lead to cell death, which is what occurs during an infarction. Without proper blood flow, the affected tissue cannot receive the necessary nutrients and oxygen to function properly.

2. How does venous block differ from arterial block in causing infarction?

Venous block differs from arterial block in causing infarction because arterial blockage occurs in the arteries, which carry oxygen-rich blood from the heart to the rest of the body. This type of blockage can cause tissue death due to lack of oxygen. Venous blockage, on the other hand, occurs in the veins, which carry oxygen-depleted blood back to the heart. This type of blockage can cause a backup of blood and fluid, leading to tissue damage and infarction.

3. Which organs are most affected by venous block causing infarction?

The organs most affected by venous block causing infarction are those with a high demand for oxygen, such as the heart, brain, and lungs. These organs have a large number of blood vessels and require a constant supply of oxygen to function properly. When a venous block occurs, the affected organ may not receive enough oxygen, leading to tissue death and potential organ failure.

4. Can venous block causing infarction be prevented?

Yes, venous block causing infarction can be prevented by maintaining a healthy lifestyle and managing any underlying conditions that may increase the risk of blockages, such as high blood pressure or high cholesterol. It is also important to seek medical attention if you experience symptoms of a venous block, such as swelling, pain, or discoloration of the affected area.

5. What are the treatment options for venous block causing infarction?

Treatment for venous block causing infarction may include medications to dissolve the blockage, surgery to remove the obstruction, or procedures to bypass the blocked vein. In some cases, lifestyle changes and management of underlying conditions may also be recommended. It is important to seek prompt medical treatment to prevent further damage and complications from a venous block.

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