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Virus mutation by artificially guided selection
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[QUOTE="artis, post: 6863215, member: 652341"] It seems based on the many random variables and time it takes and other reasons mentioned in this thread that the route labs typically chose is "gain of function" am I right? In this way one doesn't have to wait for the lucky moment one can change the virus side stepping evolution a little to make it infectious to the target cell culture to see the result. It depends on the goal of course whether they simply want to see how fast the virus will on average arrive at the needed change naturally or whether it can become infectious to certain target cells and therefore to the hosts of those cells, because it seems the latter is the result usually sought after then gain of function seems like a good avenue to find that out ? As for Covid, [USER=581757]@BillTre[/USER] refresh my memory but IIRC, before the Covid pandemic set off we had no data on any Covid or Covid like virus spreading on a large scale within the very animals you mentioned ? If we assume Covid came about naturally , it should have been present in the species that served as the intermediary (pangolins?) between the original source (bats? RatG 13?) and humans? Sure enough I doubt pre Covid anyone would have been interested to regularly test various animals for respiratory viruses within China, although assuming they did have a big database of Coronaviruses it seems likely they did routine safety checks.What does make me suspicious among other things is that Covid managed to jump species so fast and be so fit at the same time, sure respiratory viruses mutate fast but given it's capability of transmission wouldn't the likelihood that it would be seen in the intermediate animal before it jumped to humans be high? Unless of course it made no symptoms within those animals and given it couldn't be outwardly checked nobody cared enough to do blood tests for those animals. What you know [USER=581757]@BillTre[/USER] ? [/QUOTE]
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