BlackVision said:
In your study, what is the testosterone level between males and females?
That study did not refer to females. Obviously you didn't read it. But, since you ask, and since you earlier questioned the link between testosterone and obesity, here is another reference, that addresses both of those questions.
Horm Metab Res. 2003 Jun;35(6):362-6.
Sex difference in the effect of obesity on 24-hour mean serum gonadotropin levels.
Strain GW, Zumoff B, Miller LK, Rosner W.
To determine the effect of obesity on serum gonadotropin levels and any possible sex difference in the effect, we measured the 24-hour mean serum follicle-stimulating hormone (FSH) and luteinizing hormone (LH) concentrations in 62 healthy men with Body Mass Index (BMI) ranging from 20 - 94 and 61 healthy, regularly cycling women with BMIs ranging from 19 - 76. We also measured free testosterone (T) and estradiol (E2) in these subjects. There was a significant negative correlation between serum FSH and BMI in men: FSH(IU/L) = 49.9 x BMI -0.567; r = - 0.376, p = 0.0026; but a significant positive correlation between serum FSH and BMI in women: FSH(IU/L) =7.66 +/- 0.071 x BMI; r = 0.302, p = 0.018. Serum LH was weight-invariant in both sexes. In men, free T was negatively correlated with BMI: Free T (nmol/L) = 0.74 - 0.0068 x BMI; r = 0.585, p = 0.0381; and free E2 was positively correlated with BMI: Free E2 (pmol/L) = - 1.03 +/- 0.057 x BMI; r = 0.50, p = 0.0014. In obese women as a group, free T was higher than in lean women (33 +/- 6.8 S.E.M. vs. 17.4 +/- 2.0 pmol/L; p < 0.0001), and free E2 was also higher than in lean women: (6.90 +/- 0.80 vs. 4.84 +/- 0.55 pmol/L; p = 0.046). Of the many cases of hypothalamic-pituitary hormonal dysregulation that have been reported in obesity, none has been studied for sex differences. Our results mandate that possible sex differences be investigated in all cases of dysregulation.
BlackVision said:
And the reason why men have a higher homicide rate and why blacks do would probably be at least partly the same reason. The reason of course being testosterone level. Also does your study happen to mention the testosterone level of asians?
Again, it becomes obvious you are refuting a study you did not read. And you complain of Nereid's questions! I could use your tactic and tell you to do a literature search as the information must be readily available. But, here are more references for your edification.
First, a review article:
Asian J Androl. 2000 Mar;2(1):13-20.
Differences in reproductive endocrinology between Asian men and Caucasian men--a literature review.
van Houten ME, Gooren LJ.
"A later study[26], however, demonstrated that the decreased levels of 5?Á-reduced androgen metabolites in Asian men was not explained by a genetically determined low 5?Á-reductase activity. Instead, reduced levels of the androgenic ketosteroid precursors of these plasma metabolites are a more likely reason. They found a significant reduction in the production of testosterone and plasma testosterone in Chinese residents compared to Chinese migrants in Pennsylvania. No difference was found between Chinese residents and Caucasians living in Pennsylvania. No information was provided in the study regarding the amount of years for which the Chinese migrants had already resided in Pennsylvania. The study of Lookingbill DP et al, 1991[18] showed no obvious difference in serum total and free testosterone levels between Asian and Caucasian men. "
And so you know what references they are citing here, I will include those also (of course you could look them up yourself when you read the article).
[18] Lookingbill DP, Demers LM, Wang C, Leung A, Rittmaster RS, Santen RJ.Clinical and biochemical parameters of androgen action in normal healthy Caucasian versus Chinese subjects.*J Clinical Endocrinol Metab 1991; 72: 1242-8.
J Clin Endocrinol Metab. 1991 Jun;72(6):1242-8. Related Articles, Links
Stimulation of androgen-sensitive hair follicles is mediated by dihydrotestosterone (DHT), which is formed in these tissues by 5 alpha-reduction of testosterone. A possible mechanism for increased body hair in some human populations might, therefore, be an increase in 5 alpha-reductase activity, resulting in elevated tissue levels of DHT. If present, this finding could have other important clinical implications, since the 5 alpha-reductase enzyme is pivotal in the pathophysiology of prostatic disease. To explore differences in clinical and biochemical parameters of androgen action, we conducted a study of 184 caucasian and Chinese subjects in whom we evaluated chest hair density and serum levels of androgen precursors and 5 alpha-reduced androgen metabolites. Differences in chest hair density were most notable in the men, in whom comparative mean chest hair scores (using a scale of 0-4) were 3.0 vs. 0.8 (P less than 0.0001), caucasian vs. Chinese. Levels of 5 alpha-reduced androgen products were also strikingly higher in the caucasian vs. Chinese subjects. Serum 3 alpha-androstanediol glucuronide levels (nanomoles per L) were 34.7 +/- 2.4 vs. 19.7 +/- 0.9 (P less than 0.001) for the men and 21.5 +/- 3.2 vs. 9.4 +/- 0.6 (P less than 0.001) for the women, and serum levels of androsterone glucuronide (nanomoles per L) were 179 +/- 26 vs. 107 +/- 7 (P less than 0.01) for the caucasian vs. Chinese men and 173 +/- 23 vs. 81 +/- 9 (P less than 0.001) for the women. Serum levels of total and bioavailable testosterone did not differ between the racial groups, but serum levels of the precursor androgens, dehydroepiandrosterone sulfate and androstenedione, were significantly higher in the caucasian vs. Chinese men, but not in the women. We conclude that increased serum levels of 5 alpha-reduced androgen metabolites in caucasians vs. Chinese subjects provide circumstantial evidence for a racial difference in 5 alpha-reductase activity and suggest a mechanism for the increased body hair observed in the caucasian men. Increased levels of precursor androgens may also play a role.
[26] Santner SJ, Albertson B, Zhang G, Zhang GH, Santulli M, Wang C, et al. Comparative rates of androgen production and metabolism in Caucasian men and Chinese subjects.*J Clin Endocrinol Metab 1998; 83: 2104-9.
Clinically apparent prostate cancer occurs more commonly among Caucasians living in Western countries than in Chinese in the Far East. Prior studies demonstrated diminished facial and body hair and lower levels of plasma 3 alpha-androstanediol glucuronide and androsterone glucuronide in Chinese than in Caucasian men. Based upon these findings, investigators postulated that Chinese men could have diminished 5 alpha-reductase activity with a resultant decrease in prostate tissue dihydrotestosterone levels and clinically apparent prostate cancer. An alternative hypothesis suggests that decreased 3 alpha-androstanediol glucuronide and androsterone glucuronide levels might reflect reduced production of androgenic ketosteroid precursors as a result of genetic or environmental factors. The present study examined 5 alpha-reductase activity, androgenic ketosteroid precursors, and the influence of genetic and environmental/dietary factors in groups of Chinese and Caucasian men. We found no significant differences in the ratios of 5 beta-:5 alpha-reduced urinary steroids (a marker of 5 alpha-reductase activity) between Chinese subjects living in Beijing, China, and Caucasians living in Pennsylvania. To enhance the sensitivity of detection, we used an isotopic kinetic method to directly measure 5 alpha-reductase activity and found no difference in testosterone to dihydrotestosterone conversion ratios between groups. Then, addressing the alternative hypothesis, we found that the Caucasian subjects excreted significantly higher levels of individual and total androgenic ketosteroids than did their Chinese counterparts. To distinguish genetic from environmental/dietary factors as a cause of these differences, we compared Chinese men living in Pennsylvania and a similar group living in Beijing, China. We detected a reduction in testosterone production rates and total plasma testosterone and sex hormone-binding levels, but not in testosterone MCRs in Beijing Chinese as a opposed to those living in Pennsylvania. Comparing Pennsylvania Chinese with their Caucasian counterparts, we detected no significant differences in total testosterone, free and weakly bound testosterone, sex hormone-binding globulin levels, and testosterone production rates. Taken together, these studies suggest that environmental/dietary, but not genetic, factors influence androgen production and explain the differences between Caucasian and Chinese men.
Now, yet another study finds something different. The more I delve into this for you, the more I see a major problem arising...lack of repeatability. In other words, the connection is not reliable between ethnic groups and testosterone concentrations. Here, this one does show ethnic differences in serum total and free testosterone.
Cancer Epidemiol Biomarkers Prev. 1995 Oct-Nov;4(7):735-41.
Serum androgens and sex hormone-binding globulins in relation to lifestyle factors in older African-American, white, and Asian men in the United States and Canada.
Wu AH, Whittemore AS, Kolonel LN, John EM, Gallagher RP, West DW, Hankin J, Teh CZ, Dreon DM, Paffenbarger RS Jr.
"After adjustment for age and Quetelet's index, the levels of total and bioavailable testosterone were highest in Asian-Americans, intermediate in African-Americans, and lowest in whites."
If your prediction that higher levels of testosterone in a population result in increased crime/murder rates in a population, then Asian-Americans should have the highest crime/murder rate. They don't, therefore your hypothesis that it is all about testosterone must be rejected.