Is TMEM108 Effective for Treating Schizophrenia in Humans?

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Research on the TMEM108 gene indicates that mutations can impair neuronal communication, contributing to schizophrenia symptoms. A study highlights that reduced expression of TMEM108 leads to fewer and smaller dendritic spines, which are crucial for neuron communication. This finding, based on mouse models, raises questions about the potential for similar treatments in humans. The discussion emphasizes the relevance of TMEM108 research to understanding schizophrenia and the need for clarity on its implications for human treatment.
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I have found this article about schizophrenia, It talk's about TMEM working on mice, I wonder if there is similar
treatment for humans.
https://www.sciencedaily.com/releases/2017/01/170118163751.htmMutations in a gene that should enable memories and a sense of direction instead can result in imprecise communication between neurons that contributes to symptoms of schizophrenia, scientists report.

They found that dramatically reducing the amount of protein expressed by TMEM108, a gene already associated with schizophrenia, results in fewer, smaller spines, which work like communication fingers for neurons, said neuroscientist Dr. Lin Mei.
 
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